Date of Award

2010

Degree Type

Thesis

Degree Name

Master of Science

Program

Pathology

Supervisor

Dr. Joan H.M. Knoll

Second Advisor

Dr. Peter K. Rogan

Third Advisor

Dr. Ann F. Chambers

Abstract

Cancer genomes accumulate chromosomal abnormalities and gene mutations but must maintain the ability to survive in vitro. We sought evidence in breast cancer that genetic selection acts to maintain tumour survival. Analysis of genomes from 243 breast tumours revealed 766 unstable and 812 stable contiguous genomic regions. Stable DNA targets were confirmed by quantitative PCR in six breast cancer cell lines. Stable regions contained 9,463 protein coding genes, of which expression of 7,489 were analyzed. We found 78% (5804/7489) of genes had both stable copy number and expression. The breast cancer genome preferentially preserves functions that include cellular metabolic processes, gene expression regulation, DNA packaging, cellular component assembly, RNA metabolism and regulation of apoptosis. Stable genes were consistently found to be targets of therapeutic drugs. Conservation of this minimal gene set may explain the effectiveness of certain chemotherapeutic agents that act on multiple gene products in this set.

Share

COinS
 
 

To view the content in your browser, please download Adobe Reader or, alternately,
you may Download the file to your hard drive.

NOTE: The latest versions of Adobe Reader do not support viewing PDF files within Firefox on Mac OS and if you are using a modern (Intel) Mac, there is no official plugin for viewing PDF files within the browser window.