Date of Award

2011

Degree Type

Thesis

Degree Name

Master of Science

Program

Biochemistry

Supervisor

Dr. Tony Rupar

Abstract

Metachromatic leukodystrophy (MLD) is a lysosomal storage disease caused by a mutation in the enzyme, arylsulfatase A (ASA) and is characterized by progressive and fatal demyelination in the central and peripheral nervous systems. To date, no effective treatment exists and development is hampered by the blood-brain barrier (BBB). I have successfully generated fusion proteins using an ApoE fragment as a molecular Trojan horse fused to the full-length human ASA. These proteins were expressed in eukaryotic cells transduced with lentiviral expression vectors and have been assessed for in vitro enzymatic activity and kinetic properties. Although preliminary studies show that these constructs did not cross an in vitro BBB model under these experimental conditions, there is evidence to suggest that the ApoE fragment facilitates the uptake of fusion proteins into the endothelial cells of the BBB. Recommendations for further study are encouraged to reach the goal to find treatment for MLD

Share

COinS
 
 

To view the content in your browser, please download Adobe Reader or, alternately,
you may Download the file to your hard drive.

NOTE: The latest versions of Adobe Reader do not support viewing PDF files within Firefox on Mac OS and if you are using a modern (Intel) Mac, there is no official plugin for viewing PDF files within the browser window.